Reply:Â Soluble oligomers or insoluble fibrils?

Cases of familial idiopathic normal pressure hydrocephalus implicate genetic factors in disease pathogenesis.

In vivo cortical diffusion imaging relates to Alzheimer's disease neuropathology.

Associations of Sex, Race, and Apolipoprotein E Alleles With Multiple Domains of Cognition Among Older Adults.

LATE-NC risk alleles (in TMEM106B, GRN, and ABCC9 genes) among persons with African ancestry.

Apolipoprotein E isoform does not influence trans-synaptic spread of tau pathology in a mouse model.

APOE2 gene therapy reduces amyloid deposition, and improves markers of neuroinflammation and neurodegeneration in a mouse model of Alzheimer disease.

Specific detection of tau seeding activity in Alzheimer's disease using rationally designed biosensor cells.

Tau seeding and spreading in vivo is supported by both AD-derived fibrillar and oligomeric tau.

VWA3A-derived ependyma promoter drives increased therapeutic protein secretion into the CSF.

Multi-ancestry genome-wide meta-analysis of 56,241 individuals identifies LRRC4C, LHX5-AS1 and nominates ancestry-specific loci PTPRK , GRB14 , and KIAA0825 as novel risk loci for Alzheimer's disease: the Alzheimer's Disease Genetics Consortium.

Endothelial Cells Are Heterogeneous in Different Brain Regions and Are Dramatically Altered in Alzheimer's Disease.

Age-dependent increase in tau phosphorylation at serine 396 in Huntington's disease pre-frontal cortex.

Tau seeding and spreading in vivo is supported by both AD-derived fibrillar and oligomeric tau.

Interneuron diversity in the human dorsal striatum.